Paulo Jorge Gouveia Simões da Silva Oliveira


AuthID: R-000-EED

Confirmed Publications: 332



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TITLE: Dimethylaminopyridine derivatives of lupane triterpenoids are potent disruptors of mitochondrial structure and function  Full Text
AUTHORS: Jon Holy; Oksana Kolomitsyna; Dmytro Krasutsky; Paulo J Oliveira ; Edward Perkins; Pavel A Krasutsky;
PUBLISHED: 2010, SOURCE: BIOORGANIC & MEDICINAL CHEMISTRY, VOLUME: 18, ISSUE: 16
INDEXED IN: Scopus WOS CrossRef: 12
IN MY: ORCID | ResearcherID | CIÊNCIAVITAE
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TITLE: Long-term hyperglycaemia decreases gastrocnemius susceptibility to permeability transition  Full Text
AUTHORS: Jose Lumini Oliveira ; Antonio Ascensao ; Claudia V Pereira; Sergio Magalhaes; Franklin Marques ; Paulo J Oliveira ; Jose Magalhaes ;
PUBLISHED: 2010, SOURCE: EUROPEAN JOURNAL OF CLINICAL INVESTIGATION, VOLUME: 40, ISSUE: 4
INDEXED IN: Scopus WOS CrossRef: 3
IN MY: ORCID | ResearcherID | CIÊNCIAVITAE
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TITLE: Possible anti-cancer application of the phytochemicals daphnetoxin and daphnoretin- relevance of mitochondrial toxicity  Full Text
AUTHORS: Diogo, CV; Felix, L; Vilela, S; Burgeiro, A; Barbarosa, IA; Carvalho, MJ; Oliveira, PJ ; Peixoto, FP;
PUBLISHED: 2010, SOURCE: EUROPEAN JOURNAL OF CLINICAL INVESTIGATION, VOLUME: 40
INDEXED IN: WOS
IN MY: CIÊNCIAVITAE
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TITLE: Sub-chronic administration of doxorubicin to Wistar rats results in oxidative stress and unaltered apoptotic signaling in the lung  Full Text
AUTHORS: Machado, NG; Baldeiras, I ; Pereira, GC; Pereira, SP; Oliveira, PJ ;
PUBLISHED: 2010, SOURCE: CHEMICO-BIOLOGICAL INTERACTIONS, VOLUME: 188, ISSUE: 3
INDEXED IN: Scopus WOS CrossRef
IN MY: ORCID | ResearcherID | CIÊNCIAVITAE
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TITLE: Can Drug Safety be Predicted and Animal Experiments Reduced by Using Isolated Mitochondrial Fractions?
AUTHORS: Susana P Pereira; Goncalo C Pereira; Antonio J Moreno ; Paulo J Oliveira ;
PUBLISHED: 2009, SOURCE: ATLA-ALTERNATIVES TO LABORATORY ANIMALS, VOLUME: 37, ISSUE: 4
INDEXED IN: Scopus WOS
IN MY: ORCID | ResearcherID | CIÊNCIAVITAE
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TITLE: Doxorubicin-induced mitochondrial dysfunction is secondary to nuclear p53 activation in H9c2 cardiomyoblasts  Full Text
AUTHORS: Vilma A Sardao ; Paulo J Oliveira ; Jon Holy; Catarina R Oliveira ; Kendall B Wallace;
PUBLISHED: 2009, SOURCE: CANCER CHEMOTHERAPY AND PHARMACOLOGY, VOLUME: 64, ISSUE: 4
INDEXED IN: Scopus WOS
IN MY: ResearcherID
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