22
TITLE: Notch1 Is Pan-Endothelial at the Onset of Flow and Regulated by Flow  Full Text
AUTHORS: Espen D Jahnsen; Alexandre Trindade ; Hans C Zaun; Stephanie Lehoux; Antonio Duarte ; Elizabeth A V Jones;
PUBLISHED: 2015, SOURCE: PLOS ONE, VOLUME: 10, ISSUE: 4
INDEXED IN: Scopus WOS CrossRef
24
TITLE: Dll4-Notch signaling determines the formation of native arterial collateral networks and arterial function in mouse ischemia models
AUTHORS: Brunella Cristofaro; Yu Shi; Marcella Faria; Steven Suchting; Aurelie S Leroyer; Alexandre Trindade ; Antonio Duarte ; Ann C Zovein; Luisa L Iruela Arispe; Lina R Nih; Nathalie Kubis; Daniel Henrion; Laurent Loufrani; Mihail Todiras; Johanna Schleifenbaum; Maik Gollasch; Zhen W Zhuang; Michael Simons; Anne Eichmann; Ferdinand le Noble;
PUBLISHED: 2013, SOURCE: DEVELOPMENT, VOLUME: 140, ISSUE: 8
INDEXED IN: Scopus WOS CrossRef
26
TITLE: Inhibition of Notch signaling by Dll4-Fc promotes reperfusion of acutely ischemic tissues  Full Text
AUTHORS: Ren Liu; Alexandre Trindade ; Zhanfeng F Sun; Ram Kumar; Fred A Weaver; Valery Krasnoperov; Kranthi Naga; Antonio Duarte ; Parkash S Gill;
PUBLISHED: 2012, SOURCE: BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, VOLUME: 418, ISSUE: 1
INDEXED IN: Scopus WOS CrossRef
29
TITLE: Combination of Dll4/Notch and Ephrin-B2/EphB4 targeted therapy is highly effective in disrupting tumor angiogenesis  Full Text
AUTHORS: Dusan Djokovic; Alexandre Trindade ; Joana Gigante; Marina Badenes; Lilliana Silva; Ren Liu; Xiuqing Q Li; Ming Gong; Valery Krasnoperov; Parkash S Gill; Antonio Duarte ;
PUBLISHED: 2010, SOURCE: BMC CANCER, VOLUME: 10, ISSUE: 1
INDEXED IN: Scopus WOS CrossRef
30
TITLE: Loss of Notch signalling induced by Dll4 causes arterial calibre reduction by increasing endothelial cell response to angiogenic stimuli  Full Text
AUTHORS: Rui Benedito; Alexandre Trindade ; Masanori Hirashima; Domingos Henrique ; Luis Lopes da Costa ; Janet Rossant; Parkash S Gill; Antonio Duarte ;
PUBLISHED: 2008, SOURCE: BMC DEVELOPMENTAL BIOLOGY, VOLUME: 8, ISSUE: 1
INDEXED IN: Scopus WOS CrossRef
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