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Beta-Amyloid Precursor Protein Intracellular Domain Controls Mitochondrial Function by Modulating Phosphatase and Tensin Homolog-Induced Kinase 1 Transcription in Cells and in Alzheimer Mice Models

AuthID
P-00N-KMT

Author(s)

Goiran, T

Duplan, E

Chami, M

Bourgeois, A

El Manaa, W

Rouland, L

Dunys, J

Lauritzen, I

You, H

Stambolic, V

Biferi, MG

Barkats, M

Pimplikar, SW

Sergeant, N

Colin, M

Morais, VA

Pardossi Piquard, R

Checler, F

da Costa, CA

Document Type

Article

Year published

2018

Published

in BIOLOGICAL PSYCHIATRY, ISSN: 0006-3223

Volume: 83, Issue: 5, Pages: 416-427 (12)

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Publication Identifiers

DOI: 10.1016/j.biopsych.2017.04.011

Wos: WOS:000424319700006

Source Identifiers

ISSN: 0006-3223

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Beta-Amyloid Precursor Protein Intracellular Domain Controls Mitochondrial Function by Modulating Phosphatase and Tensin Homolog-Induced Kinase 1 Transcription in Cells and in Alzheimer Mice Models

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Beta-Amyloid Precursor Protein Intracellular Domain Controls Mitochondrial Function by Modulating Phosphatase and Tensin Homolog-Induced Kinase 1 Transcription in Cells and in Alzheimer Mice Models

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